Study links mTORC1 to astrocyte reactivity in tuberous sclerosis
Nature reports that reactive astrocytes emerge as a primary consequence of TSC2 loss in tuberous sclerosis complex.
TLDR
Nature reports that mTORC1 drives cell-autonomous astrocyte reactivity in tuberous sclerosis. The paper describes reactive astrocytes as a primary consequence of TSC2 loss, implicating glial dysfunction as a primary driver of the condition’s development.
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