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@pseudorius Do you only read anti-marijuana studies, or all of them?And are you aware that the studies you linked have been debunked?Cancer Improves Outcomes In Hepatitis C<a class="user" href="http://www.eurojgh.com/pt/re/ejgh/abstract.00042737-200610000-00005.htm;jsessionid=J4GWjhVclQ9rvKmJ7ZKdQrrVTTpvk1X8GwtymyJJZbl4snDPy0dv!-1997255598!181195629!8091!-1&quot; rel="nofollow">http://www.eurojgh.com/pt/re/ejgh/abstract.0004273 ...</a>Study Finds No Cancer-Marijuana Link<a class="user" href="http://www.washingtonpost.com/wp-dyn/content/article/2006/05/25/AR2006052501729_pf.html&quot; rel="nofollow">http://www.washingtonpost.com/wp-dyn/content/artic ...</a>Testicular cancer link, unproven, overstated<a class="user" href="http://stash.norml.org/going-to-pot-weed-and-testicular-cancer/&quot; rel="nofollow">http://stash.norml.org/going-to-pot-weed-and-testi ...</a>There has never been a single case of lung cancer documented to be caused by marijuana.Pot is not linked to lung cancer, even for the heaviest smokers:<a class="user" href="http://www.webmd.com/lung-cancer/news/20060523/pot-smoking-not-linked-to-lung-cancer&quot; rel="nofollow">http://www.webmd.com/lung-cancer/news/20060523/pot ...</a>Marijuana's supposed &quot;disruptive&quot; effects on the brain have been debunked:<a class="user" href="http://www.drugscience.org/Petition/C3I.html&quot; rel="nofollow">http://www.drugscience.org/Petition/C3I.html&lt;/a&gt;Marijuana only effects memory during active intoxication, not afterwards:<a class="user" href="http://www.drugtext.org/sub/marmyt1.html&quot; rel="nofollow">http://www.drugtext.org/sub/marmyt1.html&lt;/a&gt;Also:1. Baron and Folan, &quot;Ulcerative Colitis and Marijuana&quot;, Annals of Internal Medicine, Vol. 112, No.6, p 47, 1990, &quot;The symptoms of ulcerative colitis were repeatedly relieved by smoked cannabis.&quot;) 2. National Academy of Science, Institute of Medicine, &quot;Marijuana and Medicine; Assessing the Science Base &quot; Executive Summary, 1999, &quot;Conclusion: Scientific data indicate the potential therapeutic value of cannabinoid drugs, primarily THC, for pain relief, control of nausea and vomiting, and appetite stimulation;&quot;) 3. Pharmos Corporation Press Release, May 21 1998, Researchers reported that experimental treatment of rats suffering from ulcerative colitis with Dexanabinol (synthetic cannabidiol-CBD) &quot;significantly reduced the anorexia and the colonic inflammation associated with this condition compared with untreated rats.&quot;) 4. BW Healthwire, January 1998, &quot;Pre-clinical studies show CT-3 reduces chronic and acute inflammation and reduces destruction of joints.&quot; Atlantic Pharmaceuticals was evaluating CT-3, a cannabinoid derivative. The company reported &quot;In recent studies, the agent was found to reduce inflammation and prevent the destruction of joint tissue.&quot;) 5. Formukong, Evans, and Evans &quot;Analgesic and anti-inflammatory activity of constituents of cannabis sativa l&quot; Inflammation, Vol. 12 No. 4, pp361-371, 1988, &quot;Cannabidol is more effective than aspirin in reducing inflammation&quot; (Cannabidol or CBD, is one of the scores of cannibinoids found in marijuana other than THC)) 6. Holdcroft, et al., &quot;Pain relief with oral cannabinoids in familial Mediterranean fever&quot; Anesthesia, Vol. 52, No. 5, May 1997. This research paper done at Hammersmith Hospital in London confirmed cannabis' analgesic effects in the first UK clinical trial. The paper states &quot;Cannabinoids have analgesic and possibly anti-inflammatory properties but their clinical use has been restricted by legislation&quot; 7. Zeltser, R.; Seltzer, Z.; Eisen, A.; Feigenbaum, J.J.; and Mechoulam, R &quot;Suppression of neuropathic pain behavior in rats by a non-psychotropic synthetic cannabinoid with NMDA receptor-blocking properties&quot; Pain Vol. 47(1): 95-103, October. 1991, The study found cannabis surpressed neuropathic pain which complicates many CNS diseases. Cannabis was effective when few available therapies provided even partial relief.) 8. Friedman, H.; Klein, T.W.; Newton, C.; and Daaka, Y., &quot;Marijuana receptors and immunomodulation.&quot; Advances in Experimental Medicine and Biology 373: pp103-113, 1995, &quot;The study suggested that the immunosuppressive effects of cannabinoids might be useful clinically; for example, in treating multiple sclerosis.) 9. Grotenhermen Dr. Franjo, IACM-Bulletin of 25 June 2000, (IACM): &quot;We know from animal studies that THC inhibits the production of Th-1 cytokines such as IL-1, IL-2, and IFN-gamma and stimulates the production of Th-2 cytokines such as IL-4, IL-10, and TGF-beta. This would give reason for a causal therapeutic use of THC in certain autoimmune diseases that appear to be Th-1 mediated such as Crohn's disease, a form of chronic intestinal inflammation, and rheumatoid arthritis.&quot;)

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